There is strong evidence that FMCP has a hereditary component.
Loss of elbow range of motion (ROM ) is due to osteoarthritis (OA) .
The exact etiology of FMCP is unknown. Several theories have been proposed. 1) a result from a disruption of endochondral ossification of the coronoid process. 2) developmental elbow incongruity, 3) Asynchronous growth of the radius and unla, 4) a vascular anomaly , 5) trochlear notch dysplasia.
Coronoid fragmentation is characterized by separation of, fissuring of, or osteonecrosis of the process.
The end result is the development of OA / Degenerative Joint Disease.
Cartilage damage varies between patients and the specific cause is unknown. Some cartilage wear may be due to abrasion against the loose fragment, but this does not explain the severe cartilage loss in the medial compartment is some cases.
Surgical debridement is aimed at removing the loose fragment and associated pain from the “rock in the shoe “ effect.
Prognosis varies by age and degree of OA at the time of surgery. Dogs under 8 months of age most always have less progression of disease after FMCP removal than those over 8 months. This is probably due to the continued formation of hyline cartilage which ceases as the growth plates close.
Once OA develops in a joint….IT WILL PROGESS…. Whether surgical removal is performed or not.
THE BASIS FOR TREATMENT OF FMCP IS FRAGMENT REMOVAL AND LIFE LONG MEDICAL MANAGEMENT.
WEIGHT CONTROL is important. Obesity causes excessively high forces to be transmitted through an already abnormal joints. Soreness may increase and arthritic changes may be accelerated.
EXERCISE RESTRICTIONS are needed to be effective. Full out running, jumping, and off leash play should be prohibited as much as possible. Extended walks, swimming and leash jogging can be helpful to build strength and muscle mass. DOGS DO NOT KNOW THEIR LIMITS. You must take responsibility for limiting the exercise for your pet.